Fri Aug 26th, 2011 at 02:47:46 AM EST
Had an interesting conversation re the new IBM brain-style neural chip yesterday over Heineken with a Google of my acquaintance, who altho a brilliant programmer and all around heavy hitter, seems almost theologically committed to the thesis that we won't be able to build an artificial intelligence, at all, period, never: Humans are too complicated.
Well, I said, number one, why does an AI have to be like a human?
If we left out the horrible spaghetti monster of evolved processor nets for dangers we never now face, if we left out the God processor in the hypothalamus or the cingulate gyrus blah blah, and just tried build the rational processor, an Asperger's AI, how much would it take?
Hmmm.... he said, and off we went to Pasta e fagioli and less taxing blab... but I'd like to know what you think:
EDIT (of course you read /. )
"There is increasing, but largely indirect, evidence pointing to an effect of commensal gut microbiota on the central nervous system (CNS). However, it is unknown whether lactic acid bacteria such as Lactobacillus rhamnosus could have a direct effect on neurotransmitter receptors in the CNS in normal, healthy animals. GABA is the main CNS inhibitory neurotransmitter and is significantly involved in regulating many physiological and psychological processes. Alterations in central GABA receptor expression are implicated in the pathogenesis of anxiety and depression, which are highly comorbid with functional bowel disorders. In this work, we show that chronic treatment with L. rhamnosus (JB-1) induced region-dependent alterations in GABAB1b mRNA in the brain with increases in cortical regions (cingulate and prelimbic) and concomitant reductions in expression in the hippocampus, amygdala, and locus coeruleus, in comparison with control-fed mice. In addition, L. rhamnosus (JB-1) reduced GABAAα2 mRNA expression in the prefrontal cortex and amygdala, but increased GABAAα2 in the hippocampus. Importantly, L. rhamnosus (JB-1) reduced stress-induced corticosterone and anxiety- and depression-related behavior. Moreover, the neurochemical and behavioral effects were not found in vagotomized mice, identifying the vagus as a major modulatory constitutive communication pathway between the bacteria exposed to the gut and the brain. Together, these findings highlight the important role of bacteria in the bidirectional communication of the gut–brain axis and suggest that certain organisms may prove to be useful therapeutic adjuncts in stress-related disorders such as anxiety and depression."